What Makes Forest-Based Heterogeneous Treatment Effect Estimators Work?
- URL: http://arxiv.org/abs/2206.10323v2
- Date: Wed, 20 Dec 2023 14:20:51 GMT
- Title: What Makes Forest-Based Heterogeneous Treatment Effect Estimators Work?
- Authors: Susanne Dandl and Torsten Hothorn and Heidi Seibold and Erik Sverdrup
and Stefan Wager and Achim Zeileis
- Abstract summary: We show that both methods can be understood in terms of the same parameters and confounding assumptions under L2 loss.
In the randomized setting, both approaches performed akin to the new blended versions in a benchmark study.
- Score: 1.1050303097572156
- License: http://creativecommons.org/licenses/by/4.0/
- Abstract: Estimation of heterogeneous treatment effects (HTE) is of prime importance in
many disciplines, ranging from personalized medicine to economics among many
others. Random forests have been shown to be a flexible and powerful approach
to HTE estimation in both randomized trials and observational studies. In
particular "causal forests", introduced by Athey, Tibshirani and Wager (2019),
along with the R implementation in package grf were rapidly adopted. A related
approach, called "model-based forests", that is geared towards randomized
trials and simultaneously captures effects of both prognostic and predictive
variables, was introduced by Seibold, Zeileis and Hothorn (2018) along with a
modular implementation in the R package model4you.
Here, we present a unifying view that goes beyond the theoretical motivations
and investigates which computational elements make causal forests so successful
and how these can be blended with the strengths of model-based forests. To do
so, we show that both methods can be understood in terms of the same parameters
and model assumptions for an additive model under L2 loss. This theoretical
insight allows us to implement several flavors of "model-based causal forests"
and dissect their different elements in silico.
The original causal forests and model-based forests are compared with the new
blended versions in a benchmark study exploring both randomized trials and
observational settings. In the randomized setting, both approaches performed
akin. If confounding was present in the data generating process, we found local
centering of the treatment indicator with the corresponding propensities to be
the main driver for good performance. Local centering of the outcome was less
important, and might be replaced or enhanced by simultaneous split selection
with respect to both prognostic and predictive effects.
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